Company profile
Maxion Therapeutics Limited
Maxion Therapeutics | Home
Maxion Therapeutics | We are using our revolutionary KnotBody technology to create first-in-class and best-in-class protein therapeutics for treating previously untreatable ion channel- and GPCR-driven diseases.
Latest accounts
Financial period: 1 Jan 2024 to 31 Dec 2024
Company events
Reference milestones and recent Companies House filing stream events.
Accounts due
Accounts DueNext accounts due date
Confirmation statement due
Confirmation DueNext confirmation statement due date
Appoint Person Director Company With Name Date
OfficersAP01 | Transaction MzUyNzg5NjkzNGFkaXF6a2N4
Published 25 Jun 2026 08:48
Confirmation statement filed
ConfirmationLast confirmation statement made up date
Accounts With Accounts Type Small
Accounts AnalysedAA | Transaction MzUxNDEyNjAxM2FkaXF6a2N4
Published 02 Apr 2026 14:27
Accounts filed
AccountsLast accounts made up date
Capital Allotment Shares
CapitalSH01 | Transaction MzQ0NDkwODI1M2FkaXF6a2N4
Published 26 Nov 2024 10:56
Incorporated
InceptionCompany registered at Companies House
Public funding
Projects
Development of a novel class of ion channel blockers for the treatment of autoimmune disease
1 Apr 2023 to 31 Jan 2026
Autoimmune diseases such as Inflammatory Bowel Disease, Type 1 Diabetes, Rheumatoid Arthritis and Psoriasis are driven by the protective immune system attacking the body itself. These conditions are often poorly treated with existing therapeutics. Autoimmune diseases are driven by a subset of T cells (part of the body's immune system) which are uniquely d...
An Innovative Molecular Fusion Format That Targets Ion Channel-Driven Diseases
1 May 2021 to 31 Oct 2022
Maxion is a newly founded UK therapeutic development SME solving a significant unmet global need with its novel, disruptive platform that can generate antibody-like therapeutics to target ion channel-driven diseases. Taking inspiration from nature, Maxion's founders have combined ion channel modulating mini-proteins found in venom and other sources called...